Turinabol - Unit: 100 pills (10 mg/pill)

Turinabol - Unit: 100 pills (10 mg/pill)

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REF-OS-BT-TRB1-100
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Active Ingredient: 4-chlorodehydromethyltestosterone

Pack: 100 tabs (10mg/tab)

Manufacturer: Biotech

Half Life: 16 hours

Detection Time: 12 months

Anabolic Rating: 54

Androgenic Rating: 6

Dosage: male 15-100 mg per day, female 5-35 mg per day.

Common brands: Chloridehydromethyltestosterone, Turinabol, Turanabol, Turinover, Tbol, Turanabolic, Chlorodemethyltestosterone, Turobol, Turabol, Turinasim, Turinabolos, Exemestane, Aromex, Turinadex, Turinabolan, Turanaplex, T-Bolic, TurinoX, Turabolone, Turbobol, X-Turbol, Oral-Turinabol, Turanaxyl, GP Turan, Turanox, Methyl-1-Testosteron, Turinotabs

Turinabol®, brand of Chlorodehydromethyltestosterone tablets, is an anabolic steroid, a synthetic derivative of testosterone. Each tablet contains 10 mg of Chlorodehydro-methyltestosteron, micronized grade. It is designated chemically as 4-chloro-17b-hydroxy-17a-methyl-androst-1,4-dien-3-one. It occurs as white or practically white, odourless, crystalline powder. Practically insoluble in water; sparingly soluble in alcohol; slightly soluble in chloroform.

Each tablet also contains lactose monohydrate, sodium starch glycolate, polyvidone 25,000, microcrystalline cellulose and magnesium stearate as excipients and also contains yellow ferric oxide (El72) and indigo carmine aluminium lake (El32) as colouring agent.

CLINICAL PHARMACOLOGY:

Endogenous androgens are responsible for normal growth and development of the male sex organs and for maintenance of secondary sex characteristics. These effects include growth and maturation of the prostate, seminal vesicles, penis, and scrotum; development of male hair distribution, such as beard, pubic, chest, and axillary hair; laryngeal enlargement, vocal cord thickening, and alterations in body musculature and fat distribution. Drugs in this class also cause retention of nitrogen, sodium, potassium, and phosphorus, and decreased urinary excretion of calcium.

Androgens have been reported to increase protein anabolism and decrease protein catabolism. Nitrogen balance is improved only when there is sufficient intake of calories and protein. Androgens are responsible for the growth spurt of adolescence and for eventual termination of linear growth, brought about by fusion of the epiphyseal growth centers. In children, exogenous androgens accelerate linear growth rates, but may cause disproportionate advancement in bone maturation. Use over long periods may result in fusion of the epiphyseal growth centers and termination of the growth process. Androgens have been reported to stimulate production of red blood cells by enhancing production of erythropoietic stimulation factor.

During exogenous administration of androgens, endogenous testosterone release is inhibited through feedback inhibition of pituitary luteinizing hormone (LH). At large doses of exogenous androgens, spermatogenesis may also be suppressed through feedback inhibition of pituitary follicle stimulating hormone (FSH). Inactivation of testosterone occurs primarily in the liver.The half-life of Chlorodehydro-methyltestosteron after oral administration is approximately16 hours.

INDICATIONS:

1. schizophrenia (acute and chronic) and other psychotic states with productive and / or negative symptoms;

2. affective disorders in a variety of mental illnesses;

3. behavioral disturbances in patients with dementia with aggressive manifestation of symptoms (angry outbursts, physical violence), disorders of mental activity (agitation, delirium) or psychotic symptoms;

4. as adjuvant therapy in the treatment of mania in bipolar disorder;

5. as adjunctive therapy of behavior disorders in adolescents from 15 years and adult patients with reduced intellectual level or mental retardation, oral-turinabol in cases where destructive behavior (aggressiveness, impulsivity, autoaggression) is leading the clinical picture of the disease.

DRUG ABUSE AND DEPENDENCE:

Controlled Substance Class: Chlorodehydromethyltestosteron is a controlled substance under the Anabolic Steroids Control Act, and Turinabol ® has been assigned to Schedule III.

CONTRAINDICATIONS:

1. Dehydration and hypovolemia

2. cerebrovascular accidents

3. Parkinson's disease

4. seizures (including history)

5. severe renal or hepatic insufficiency (see. dosing recommendations)

1. drug abuse or drug dependency (see. for dosage recommendations)

2. conditions that predispose to the development of tachycardia type

3. brain tumor, intestinal obstruction, acute cases of drug overdose, Reye's syndrome (an antiemetic effect of risperidone may mask the symptoms of these conditions)

WARNINGS:

LIVER CELL TUMORS ARE REPORTED. MOST OFTEN THESE TUMORS ARE BENIGN AND ANDROGEN DEPENDENT, BUT FATAL MALIGNANT TUMORS HAVE BEEN REPORTED. WITH DRAWAL OF DRUG OFTEN RESULTS IN REGRESSION OR CESSATION OF PROGRESSION OF THE TUMOR. HOWEVER, HEPATIC TUMORS ASSOCIATED WITH ANDROGENS OR ANABOLIC STEROIDS ARE MUCH MORE VASCULAR THAN OTHER HEPATIC TUMORS AND MAY BE SILENT UNTIL LIFE-THREATENING INTRA-ABDOMINAL HEMORRHAGE DEVELOPS.

PELIOSIS HEPATIS, A CONDITION ARE ALSO REPORTED IN WHICH LIVER AND SOMETIMES SPLENIC TISSUE IS REPLACED WITH BLOOD-FILLED CYSTS, HAS BEEN REPORTED IN PATIENTS RECEVING ANDROGENIC ANABOLIC STEROID THERAPY. THESE CYSTS ARE SOMETIMES PRESENT WITH MINIMAL HEPATIC DYSFUNCTION, BUT AT OTHER TIMES THEY HAVE BEEN ASSOCIATED WITH LIVER FAILURE. THEY ARE OFTEN NOT RECOGNIZED UNTIL LIFE-THREATENING LIVER FAILURE OR INTRA-ABDOMINAL HEMORRHAGE DEVELOPS. WITHDRAWAL OF DRUG USUALLY RESULTS IN COMPLETE DISAPPERRANCE OF LESIONS.

BLOOD LIPID CHANGES THAT ARE KNOWN TO BE ASSOCIATED WITH INCREASED RISK OF ATHEROSCLEROSIS ARE SEEN IN PATIENTS TREATED WITH ANDROGENS AND ANABOLIC STEROIDS. THESE CHANGES INCLUDE DECREASED HIGH-DENSITY LIPOPROTEIN AND SOMETIMES INCREASED LOW-DENSITY LIPOPROTEIN. THE CHANGES MAY BE VERY MARKED AND COULD HAVE A SERIOUS IMPACT ON THE RISK OF ATHEROSCLEROSIS AND CORONARY ARTERY DISEASE.

Hypercalcemia may occur in immobilized patients and in patients with breast cancer. If this occurs, the drug should be discontinued.

Prolonged use of high doses of androgens (principally the 17-a alkyl-androgens) has been associated with development of hepatic adenomas, hepatocellular carcinoma, and peliosis hepatis—all potentially life-threatening complications.

Cholestatic hepatitis and jaundice may occur with 17-a-alkyl-androgens. Should this occur, the drug should be discontinued. This is reversible with discontinuation of the drug.

Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

Edema, with or without congestive heart failure, may be a serious complication in patients with pre-existing cardiac, renal or hepatic disease.

Gynecomastia may develop and occasionally persists in patients being treated for hypogonadism.

Androgen therapy should be used cautiously in males with delayed puberty. Androgens can accelerate bone maturation without producing compensatory gain in linear growth. The effect on bone maturation should be monitored by assessing bone age of the wrist and hand every six months.

This drug has not been shown to be safe and effective for the enhancement of athletic performance. Because of the potential risk of serious adverse health effects, this drug should not be used for such purpose.

ANABOLIC STEROIDS HAVE NOT BEEN SHOWN TO ENHANCE ATHLETIC ABILITY.

PRECAUTIONS:

General

Women should be observed for signs of virilization which is usual following androgen use at high doses. Discontinuation of drug therapy at the time of evidence of mild virilism is necessary to prevent irreversible

virilization. A decision may be made by the patient and the physician that some virilization will be tolerated during treatment for breast carcinoma.

Patients with benign prostatic hypertrophy may develop acute urethral obstruction. Priapism or excessive sexual stimulation may develop. Oligospermia may occur after prolonged administration or excessive dosage. If any of these effects appear, the androgen should be stopped and if restarted, a lower dosage should be utilized.

This product contains FD&C which may cause allergic type reactions (including bronchial asthma) in certain susceptible individuals. Although the overall incidence of FD&C sensitivity in the general population is low, it is frequently seen in patients who also have aspirin hypersensitivity.

Information for patients

Patients should be instructed to report any of the following: nausea, vomiting, changes in skin color, and ankle swelling. Males should be instructed to report too frequent or persistent erections of the penis and females any hoarseness, acne, changes in menstrual periods or increase in facial hair.

Carcinogenesis, mutagenesis, impairment of fertility

Animal data: Testosterone has been tested by subcutaneous injection and implantation in mice and rats. The implant induced cervical-uterine tumors in mice, which metastasized in some cases. There is suggestive evidence that injection of testosterone into some strains of female mice increases their susceptibility to hepatoma. Testosterone is also known to increase the number of tumors and decrease the degree of differentiation of chemically- induced carcinomas of the liver in rats.

Human data: There are rare reports of hepatocellular carcinoma in patients receiving longterm therapy with androgens in high doses. Withdrawal of the drugs did not lead to regression of the tumors in all cases.

Geriatric patients treated with androgens may be at an increased risk of developing prostatic hypertrophy and prostatic carcinoma although conclusive evidence to support this concept is lacking.

This compound has not been tested for mutagenic potential. However, as noted above, carcinogenic effects have been attributed to treatment with androgenic hormones. The potential carcinogenic effects likely occur through a hormonal mechanism rather than by a direct chemical interaction mechanism.

Impairment of fertility was not tested directly in animal species. However, as noted below under Adverse Reactions, oligospermia in males and amenorrhea in females are potential adverse effects of treatment with Turinabol*. Therefore, impairment of fertility is a possible outcome of treatment with Turinabol®.

Pregnancy

Teratogenic effects: Pregnancy Category X. (See CONTRAINDICATIONS.)

Nursing mothers

Turinabol® is not recommended for use in nursing mothers.

Pediatric use

Androgen therapy should be used very cautiously in children and only by specialists aware of the adverse effects on bone maturation. Skeletal maturation must be monitored every six months by an X-ray of the hand and wrist (See WARNINGS).

ADVERSE REACTIONS:

Endocrine and urogenital:

Female: the most common side effects of androgen therapy are amenorrhea and other menstrual irregularities; inhibition of gonadotropin secretion; and virilization, including deepening of the voice and clitoral enlargement. The latter usually is not reversible after androgens arc discontinued. When administered to a pregnant woman, androgens can cause virilization of external genitalia of the female fetus.

Male: Gynecomastia, and excessive frequency and duration of penile erections. Oligospermia may occur at high dosage.

Skin and appendages: Hirsutism, male pattern of baldness, seborrhea, and acne.

Fluid and electrolyte disturbances: Retention of sodium, chloride, water, potassium, calcium, and inorganic phosphates.

Gastrointestinal: Nausea, cholestatic jaundice, alterations in liver function tests, rarely hepatocellular neoplasms and peliosis hepatis (See WARNINGS).Hematologic: Suppression of clotting factors II, V, VII, and X, bleeding in patients on concomitant anticoagulant therapy, and polycythemia.

Nervous system: Increased or decreased libido, headache, anxiety, depression, and generalized paresthesia.

Allergic: Hypersensitivity, including skin manifestations and anaphylactoid reactions.

OVERDOSAGE:

There have been no reports of acute overdosage with the androgens.

DRUG INTERACTION:

Androgens may increase sensitivity to oral anticoagulants. Dosage of the anticoagulant may require reduction in order to maintain satisfactory therapeutic hypoprothrombinemia. Concurrent administration of oxyphenbutazone and androgens may result in elevated serum levels of oxyphenbutazone.

In diabetic patients, the metabolic effects of androgens may decrease blood glucose and, therefore, insulin requirements.

PATIENT MONITORING:

Laboratory tests

Women with disseminated breast carcinoma should have frequent determination of urine and serum calcium levels during the course of androgen therapy (See WARNINGS).

Because of the hepatotoxicity associated with the use of 17-alpha-alkylated androgens, liver function tests should be obtained periodically. Periodic (every six months) X-ray examinations of bone age should be made during treatment of prepubertal males to determine the rate of bone maturation and the effects of androgen therapy on the epiphyseal centers.

Hemoglobin and hematocrit levels (to detect polycythemia) should be checked periodically in patients receiving long term androgen administration.

Serum cholesterol may increase during androgen therapy.

Drug/Laboratory test interferences

Androgens may decrease levels of thyroxine-binding globulin, resulting in decreased total T4 serum levels and increased resin uptake of T3 and T4. Free thyroid hormone levels remain unchanged, however, and there is no clinical evidence of thyroid dysfunction.

Lipid profile: Serum Cholesterol, HDL, LDL, TG.

Hemoglobin and Hematocrit,

Liver function test: Total protein. Albumin, Globulin, Total and direct bilirubin, AST, ALT and alkaline phosphatase, tumor marker for liver: AFP and CA19-9 Prostatic specific antigen: PSA, Testosterone: total, free, and bioavailable. Dihydrotestosterone & Estradiol

Male patients over 40 should undergo a digital rectal examination and evaluate PSA prior to androgen use. Periodic evaluations of the prostate should continue while on androgen therapy, especially in patients with difficulty in urination or with changes in voiding habits.

DOSAGE AND ADMINISTRATION:

The dosage will vary depending upon the individual, the condition being treated, and its severity. The total daily oral dose may be administered single or in divided (three or four) doses.

Oral-turinabol for sucking are fragile, they should not be forced through the foil packaging, as they can break. The package is opened by gently pulling the edge of the blister foil, marked by a point and remove the tablet, and then it should be put into the language immediately. Tablet turnibol begins to disperse in the mouth within seconds and can be swallowed without water and mix the drug in the mouth with food. Tablets should not be chewed or crack. Schizophrenia. Adults and children over 15 years.

Risperidone can be administered once or twice a day. Initial dose – 2 mg per day. On the second day, the dose should be increased to 4 mg per day. From this point the dose can either maintain the same level, either individually adjusted if necessary. Typically, the optimal dose is 4.6 mg per day. In some cases it may be justified by a slower increase in the dose and lower initial and maintenance doses. Doses greater than 10 mg per day did not show a higher efficiency compared to lower doses and may cause the appearance of extrapyramidal symptoms. Due to the fact that the safety of doses above 16 mg per day has not been studied, doses above this level can not be used. For information on the use of a drug for the treatment of schizophrenia oral-turinabol in children younger than 15 years no. Elderly patients. It is recommended methandienone initial dose of 0.5 mg per administration twice a day . The dosage can be individually increased by 0.5 mg twice per day to 1.2 mg twice a day. Liver and kidney diseases. It is recommended initial dose of 0.5 mg to receiving two times a day. This dose can be gradually increased to 1-2 mg twice daily intake.Abuse of drugs or drug dependency – the recommended daily dose -. 2-4 mg . Behavioral disorders in patients with dementia is recommended that the initial dose of 0.25 mg per reception twice daily (adequate dosage form should be used). If necessary dosage can be individually increased by 0.25 mg 2 times a day, no more than a day. For most patients, the optimal dose is 0.5 mg twice a day. However, some patients are shown receiving 1 mg 2 times a day. On reaching the optimum dose of the drug can be recommended once daily.

HOW SUPPLIED:

Turinabol® 10 mg is supplied in bottle of 100 green tablets.

For shelf-life please refer to the imprint on the pack.

Keep out of reach of children.

Should be at controlled room temperatures 15-30°C (59-86°F)

Protect from sun light

This drug has not been shown to be safe and effective for the enhancement of athletic performance!

1 Reviews

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I used turinabol in a few cycles and it worked great for me. thank you.
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